Author

F. Terzi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:mouse· 4 Research Focus:cell signaling & trafficking· 2 Research Focus:development & morphogenesis· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:cancer & tumor microenvironment· 1 Organism:zebrafish· 1

Frequent Co-Authors

A. Aka· 2 shared A. Viau· 2 shared C. Nguyen· 2 shared E. W. Kuehn· 2 shared F. Bienaime· 2 shared M. Burtin· 2 shared A. Claude-Taupin· 1 shared A. Regnier· 1 shared

Activity by Year

2019-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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2 shared papersPhysiology

A. Aka

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Cilia to basement membrane signalling is a biomechanical driver of autosomal dominant polycystic kidney disease

M. Mazloum, B. Lapin, A. Viau, R. Alghamdi, M. Burtin, P. Houillier, L. Cheval, G. Crambert, A. Aka, E. W. Kuehn, C. Cohen, S. Descroix, T. Busch, M. Koettgen, S. Garbay, , F. Bienaime

This study identifies biomechanical changes in the tubular basement membrane as drivers of cyst formation in ADPKD, linking cilia and polycystin-1 function.

bioRxiv · Jun 6, 2024 · PreprintPhysiology

Lcn2 protects from cisplatin induced AKI by regulating DNA damage response via EGFR activation

A. Zablocki, E. Marques, L. Yammine, C. Nguyen, F. Terzi, M. Gallazzini

This study shows Lcn2 protects kidneys from cisplatin-induced AKI by activating the EGFR pathway to reduce apoptosis.

bioRxiv · May 3, 2024 · PreprintCell Biology

YAP-dependent autophagy is controlled by AMPK, SIRT1 and flow intensity in kidney epithelial cells.

A. Claude-Taupin, F. Roccio, M. Garfa-Traore, A. Regnier, M. Burtin, E. Morel, F. Terzi, P. Codogno, N. Dupont

The paper shows YAP/TAZ inhibits autophagy in kidney epithelial cells under fluid flow, mediated by AMPK and SIRT1, and links this to CKD progression.

bioRxiv · Jan 9, 2023Cell Biology

Tubular STAT3 limits renal inflammation in autosomal dominant polycystic kidney disease

A. Viau, M. Baziz, A. Aka, C. Nguyen, E. W. Kuehn, F. Terzi, F. Bienaime

This study shows that STAT3 limits renal inflammation in ADPKD by repressing inflammatory chemokines, despite not driving cyst growth.

bioRxiv · Dec 12, 2019Pathology