Author

Anaïs Vermare

Centre National de la Recherche Scientifique

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell-cell communication· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1 Assay:imaging· 1 Organism:mouse· 1

Frequent Co-Authors

Emmanuel DonnadieuiD· 2 shared Frédéric PendinoiD· 2 shared Luca SimulaiD· 2 shared Nadège BercoviciiD· 2 shared Adrien RouaultiD· 1 shared Alexandre BoissonnasiD· 1 shared Andrea ValentiniiD· 1 shared Annavera VenturaiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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2 shared papersImmunology

Emmanuel DonnadieuORCID

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2 papers

Recent·Most cited

Monocytes promote intraepithelial infiltration of effector memory CD8+ T cells in regressing tumors

Anaïs Vermare, Annavera Ventura, Adrien Rouault, Luca Simula, Malvina Seradj, Leila Lhuillier, Eléonore Weber-Delacroix, Kevin Mulder, Lene Vimeux, David Espie, Karine Bailly, Brigitte Izac, Benjamin Saintpierre, Wail Zeitouni, Ariane Jolly, , Nadège Bercovici

The paper identifies monocyte interactions with CD8+ T cells during tumor regression, showing their role in promoting T-cell infiltration and anti-tumor immunity.

bioRxiv · May 29, 2026 · PreprintImmunology

An in vivo CRISPR screen unveils top target genes to improve CAR-T cell efficacy in a solid tumor model

Mattia Fumagalli, Dongjie An, Luca Simula, Camille Combe, Lisa Aziez, Yannick Simoni, Marie‐Clotilde Alves‐Guerra, Andrea Valentini, Maude Marchais, Anaïs Vermare, Josquin Moraly, Simona Manni, Maria Cecilia Quadraccia, Concetta Quintarelli, Biagio De Angelis, , Frédéric Pendino

This study uses an in vivo CRISPR screen to identify genes that enhance CAR-T cell efficacy in a solid tumor model, finding that disrupting REGNASE-1, SOCS1, PTPN2, and P16/NK4A improves tumor eradication.

Molecular Therapy · Apr 4, 2026Peer-reviewedImmunology