Author

Ann F. Gilliat

University College London

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Developmental Biology· 1 Genetics· 1 Physiology· 1

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Organism:C. elegans· 3 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Approach:machine learning (classical)· 1

Frequent Co-Authors

David GemsiD· 3 shared Marina EzcurraiD· 3 shared Alexandre BenedettoiD· 2 shared Catherine AuiD· 2 shared Hongyuan WangiD· 2 shared Aihan ZhangiD· 1 shared Alexandra L. Petrache· 1 shared Carina C. KerniD· 1 shared

Activity by Year

2017-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 32 mutual co-author links

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3 shared papersGenetics

David GemsORCID

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3 papers

Recent·Most cited

Machine learning predicts lifespan and underlying causes of death in aging C. elegans

Carina C. Kern, Petru Manescu, Matt Cuffaro, Catherine Au, Aihan Zhang, Hongyuan Wang, Ann F. Gilliat, Marina Ezcurra, David Gems

The paper uses machine learning to predict lifespan and causes of death in aging C. elegans by analyzing senescent pathology and genetic factors.

bioRxiv · Mar 20, 2024 · PreprintGenetics

Monsters in the uterus: A parthenogenetic quasi-program causes teratoma-like tumors during aging in wild-type C. elegans

Hongyuan Wang, Yuan Zhao, Marina Ezcurra, Alexandre Benedetto, Ann F. Gilliat, Josephine E. E. U. Hellberg, Ziyu Ren, Evgeniy R. Galimov, Trin Athigapanich, Johannes Girstmair, Maximilian J. Telford, Colin T. Dolphin, Zhizhou Zhang, David Gems

The paper investigates uterine tumors in C. elegans as a senescent pathology caused by quasi-programs, showing similarities to teratomas and senescence.

npj Aging and Mechanisms of Disease · Jun 7, 2018Peer-reviewedDevelopmental Biology

Autophagy-dependent gut-to-yolk biomass conversion generates visceral polymorbidity in aging C. elegans

Marina Ezcurra, Alexandre Benedetto, Thanet Sornda, Ann F. Gilliat, Catherine Au, Qifeng Zhang, Sophie van Schelt, Alexandra L. Petrache, Yila de la Guardia, Shoshana Bar-Nun, Eleanor Tyler, Michael J. Wakelam, David Gems

This study identifies autophagy-mediated gut-to-yolk biomass conversion as a cause of visceral pathologies and polymorbidity in aging C. elegans, and shows that inhibiting this process extends lifespan.

bioRxiv · Dec 15, 2017 · PreprintPhysiology