Author

Anna Klingseisen

Multiple Sclerosis Society

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Neuroscience· 2

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Approach:experimental assay· 2 Organism:zebrafish· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:human· 1 Assay:imaging· 1 Research Focus:spatial domain detection· 1 Assay:spatial transcriptomics· 1

Frequent Co-Authors

David Anthony LyonsiD· 2 shared Jason J. EarlyiD· 2 shared Anna WilliamsiD· 1 shared J WilliamsoniD· 1 shared Jenea M. BiniD· 1 shared Katy LH Marshall-PhelpsiD· 1 shared Lida ZoupiiD· 1 shared Linde KegeliD· 1 shared

Activity by Year

2019-2022 · peak 1/yr

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10 top collaborators · 30 mutual co-author links

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2 shared papersNeuroscience

David Anthony LyonsORCID

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2 papers

Recent·Most cited

New oligodendrocytes exhibit more abundant and accurate myelin regeneration than those that survive demyelination

Sarah A. Neely, J Williamson, Anna Klingseisen, Lida Zoupi, Jason J. Early, Anna Williams, David Anthony Lyons

The paper compares remyelination by surviving and newly generated oligodendrocytes in MS, finding that newly generated cells produce more accurate and abundant myelin sheaths.

Nature Neuroscience · Feb 14, 2022Peer-reviewedNeuroscience

Disruption to NKCC1 impairs the response of myelinating Schwann cells to neuronal activity and leads to severe peripheral nerve pathology

Linde Kegel, Katy LH Marshall-Phelps, Marion Baraban, Rafael Almeida, Maria Rubio-Brotons, Anna Klingseisen, Silvia K. Benito-Kwiecinski, Jason J. Early, Jenea M. Bin, Matthew R. Livesey, Richard J. Poole, David Anthony Lyons

This study shows that disrupting NKCC1 in Schwann cells impairs their response to neuronal activity, leading to myelin defects and nerve pathology, which can be reversed by TTX treatment.

bioRxiv · Sep 4, 2019 · PreprintNeuroscience