Author

B. Kelly

Cardiff University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:metabolomics· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

A. Hackl· 1 shared A. L. Dube· 1 shared A. M. Kabat· 1 shared A. Regina· 1 shared A.-M. Globig· 1 shared Alvaro MurilloiD· 1 shared Anne RosseriD· 1 shared B. Bengsch· 1 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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1 shared paperImmunology

A. Hackl

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Differences in white matter detected by ex vivo 9.4T MRI are associated with axonal changes in the R6/1 model of Huntington's Disease

Chiara Casella, B. Kelly, Alvaro Murillo, I. Mills-Smith, GD Parker, Chris von Ruhland, Y.A. Syed, V. Dion, C. Metzler-Baddeley, Anne Rosser, Derek Kenton Jones, Mariah J. Lelos

The paper uses MRI and histological analyses to show axonal and myelin changes in a Huntington's Disease mouse model, identifying FR as a potential MRI biomarker for WM microstructure changes.

Neurobiology of Disease · Feb 12, 2026Peer-reviewedNeuroscience

Prostaglandin E2 controls the metabolic adaptation of T cells to the intestinal microenvironment

M. Villa, D. E. Sanin, P. Apostolova, M. Corrado, A. M. Kabat, C. Cristinzio, A. Regina, G. Carrizo, N. Rana, M. A. Stanczak, F. Baixauli, K. M. Grzes, J. Cupovic, F. Solagna, A. Hackl, , E. L. Pearce

This study identifies a PGE2-autophagy-glutathione axis that enables CD8+ T cells to adapt metabolically to the intestinal microenvironment.

bioRxiv · Mar 13, 2023 · PreprintImmunology

A cell cycle-linked mechanism for the glutamine driven establishment of stem cell fate

M. J. Xiao, C.-H. Wu, G. Meek, B. Kelly, L. E. A. Young, S. Martire, D. B. Castillo, P. Saha, A. L. Dube, M. Gentry, L. A. Banaszynski, R. C. Sun, C. KIKANI

This study identifies a mechanism by which mitochondrial glutamine metabolism links cell cycle progression to stem cell fate decisions through PASK and WDR5 interactions.

bioRxiv · Mar 24, 2022Cell Biology