Author

C. C. Ebmeier

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Evolutionary Biology· 2 Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Assay:bulk RNA-seq· 2 Organism:E. coli· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

S. D. Copley· 2 shared A. B. Morgenthaler· 1 shared A. M. Whiteley· 1 shared C. Crocini· 1 shared C. I. Lau· 1 shared C. M. Walsh· 1 shared D. J. Snyder· 1 shared D. R. Hunt· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 12 mutual co-author links

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2 shared papersEvolutionary Biology

S. D. Copley

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4 papers

Recent·Most cited

A Conserved Mechanism of Cardiac Hypertrophy Regression through FoxO1

T. G. Martin, D. R. Hunt, S. J. Langer, Y. Tan, C. C. Ebmeier, C. Crocini, E. Chung, L. A. Leinwand

This study identifies FoxO1-dependent autophagy as a conserved mechanism for regression of physiological cardiac hypertrophy across species.

bioRxiv · Jan 27, 2024Physiology

The transcriptomic and proteomic ramifications of segmental amplification

R. K. Fritts, C. C. Ebmeier, S. D. Copley

This study examines the transcriptomic and proteomic effects of segmental amplification in E. coli, finding that mRNA scales with copy number but protein expression does not, and that amplifications affect non-amplified genes.

bioRxiv · Nov 21, 2023 · PreprintEvolutionary Biology

UBQLN2 restrains the domesticated retrotransposon PEG10 to maintain neuronal health in ALS

H. H. Black, J. E. Roberts, S. N. Leslie, W. Campodonico, C. C. Ebmeier, C. I. Lau, A. M. Whiteley

This study identifies UBQLN2's role in restraining the retrotransposon PEG10 to prevent neuronal damage in ALS by regulating gene expression.

bioRxiv · Mar 25, 2022Cell Biology

Mutations that improve the efficiency of a weak-link enzyme are rare compared to adaptive mutations elsewhere in the genome

A. B. Morgenthaler, W. R. Kinney, C. C. Ebmeier, C. M. Walsh, D. J. Snyder, V. S. Cooper, W. M. Old, S. D. Copley

The paper shows that mutations improving a weak-link enzyme's efficiency are rare compared to other adaptive mutations in the genome during bacterial evolution.

bioRxiv · Apr 30, 2019Evolutionary Biology