Author

C. F. Ware

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 1 Research Focus:development & morphogenesis· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. K. Lam· 1 shared A. K. Tersteegen· 1 shared A. Rajaraman· 1 shared A. Sukhina· 1 shared D. Degrandi· 1 shared E. C. Butcher· 1 shared G. Brar· 1 shared I. R. Dunay· 1 shared

Activity by Year

2020-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 12 mutual co-author links

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Curved chords = mutual co-authorship

1 shared paperMicrobiology

A. K. Lam

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Lymphotoxin β receptor: A crucial role in innate and adaptive immune responses against Toxoplasma gondii

A. K. Tersteegen, U. R. Sorg, R. Virgen-Slane, M. Helle, P. Petzsch, I. R. Dunay, K. Köhrer, D. Degrandi, C. F. Ware, K. Pfeffer

This study shows that the Lymphotoxin β receptor is critical for immune responses against Toxoplasma gondii, affecting cytokine regulation and adaptive immunity.

bioRxiv · Jan 11, 2021Immunology

Development of follicular dendritic cells in lymph nodes depends on retinoic acid mediated signaling

J. J. Koning, A. Rajaraman, R. M. Reijmers, T. Konijn, J. Pan, C. F. Ware, E. C. Butcher, R. E. Mebius

This study shows retinoic acid signaling is essential for generating follicular dendritic cells in lymph nodes, while blocking lymphotoxin signaling prevents fibroblastic reticular cell development.

bioRxiv · May 20, 2020Immunology

Deletion of immune evasion genes provides an effective vaccine design for tumor-associated herpesviruses

G. Brar, N. A. Farhat, A. Sukhina, A. K. Lam, Y. H. Kim, T. Hsu, L. Tong, W. W. Lin, C. F. Ware, M. A. Blackman, R. Sun, T.-T. Wu

This paper develops a herpesvirus vaccine by deleting immune evasion genes to enhance immunogenicity and safety, showing durable protection against latency in a mouse model.

bioRxiv · Apr 13, 2020Microbiology