Author

C. Scholtes

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

V. Giguere· 2 shared A. Blair· 1 shared A. Grubenmann· 1 shared A. Lankenau· 1 shared A. T. Hamilton· 1 shared B. Scott· 1 shared B. Testoni· 1 shared C. Charre· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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2 shared papersCell Biology

V. Giguere

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Identification of a chromatin-bound ERRα interactome network in mouse liver

C. Scholtes, C. R. Dufour, E. Pleynet, S. Kamyabiazar, P. Hutton, R. Baby, C. Guluzian, V. Giguere

The paper identifies 48 new transcriptional interactors of ERRα in mouse liver, including HCFC1, revealing a regulatory network controlling oxidative phosphorylation.

bioRxiv · Jan 2, 2024Cell Biology

Performance of the cobas(R) HBV RNA Automated Investigational Assay for the Detection and Quantification of Circulating HBV RNA in Chronic HBV Patients

C. SCHOLTES, A. T. Hamilton, M.-L. Plissonnier, C. Charre, B. Scott, L. Wang, F. Berby, J. French, B. Testoni, A. Blair, M. Subic, M. Hoppler, A. Lankenau, A. Grubenmann, M. Levrero, , F. Zoulim

This study evaluates the cobas(R) HBV RNA assay's performance in detecting and quantifying circulating HBV RNA in chronic HBV patients, showing high sensitivity and minimal interference from HBV DNA.

bioRxiv · Mar 9, 2022Microbiology

Cholesterol biosynthetic pathway induces cellular senescence through ERRa

D. V. Ziegler, M. Vernier, J. Czarnecka-Herok, C. Scholtes, C. Machon, J. Guitton, J. Rieusset, V. Giguere, N. Martin, D. Bernard

This study shows the mevalonate pathway promotes cellular senescence via ERRα, causing mitochondrial dysfunction, ROS, DNA damage, and p53-dependent senescence.

bioRxiv · Oct 20, 2021Cell Biology