Author

C. Speers

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Assay:spatial transcriptomics· 1

Frequent Co-Authors

A. de Carvalho· 1 shared A. J. Davis· 1 shared A. Rao· 1 shared A. Rehemtulla· 1 shared B. Movsas· 1 shared D. Pratt· 1 shared D. Wahl· 1 shared E. Walker· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

A. de Carvalho

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2 papers

Recent·Most cited

BUB1 regulates non-homologous end joining pathway to mediate radioresistance in triple-negative breast cancer

S. Sriramulu, S. Thoidingjam, W.-M. Chen, O. Hassan, F. Siddiqui, S. L. Brown, B. Movsas, M. D. Green, A. J. Davis, C. Speers, E. Walker, S. Nyati

This study identifies BUB1 as a regulator of non-homologous end joining (NHEJ) in triple-negative breast cancer (TNBC), showing that its inhibition sensitizes TNBC cells to radiotherapy.

bioRxiv · May 7, 2024Cancer Biology

Subclonal evolution and expansion of THY1-positive cells is associated with recurrence in glioblastoma

W. N. Al-Holou, H. Wang, V. Ravikumar, M. Oneka, R. Verhaak, H. Kim, D. Pratt, S. Camelo-Piragua, C. Speers, D. Wahl, S. Shankar, T. Hollon, O. Sagher, J. Heth, K. Muraszko, , A. Rehemtulla

This paper identifies THY1-positive, mesenchymal/stem-like cells as drivers of glioblastoma recurrence and shows that targeting TGFβ signaling can restore sensitivity to therapy.

bioRxiv · Sep 10, 2021Cancer Biology