Author

Camila Gaio

University of Calgary

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:CITE-seq· 1 Approach:experimental assay· 1 Assay:flow cytometry· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1

Frequent Co-Authors

Ben Perks· 1 shared Bruna Araújo DavidiD· 1 shared Chris TiesseniD· 1 shared Ehud Inbar· 1 shared Leah S. HohmaniD· 1 shared Matheus Batista Heitor CarneiroiD· 1 shared Matthias MackiD· 1 shared Nathan C. PetersiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperImmunology

Ben Perks

Mutual co-authors in cluster

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1 paper

Recent·Most cited

Pre-existing Th1 immunity is abrogated by ongoing recruitment of monocytic host cells that are refractory to activation

Matheus Batista Heitor Carneiro, Santiago Aguiar Espellet Soares, Chris Tiessen, Camila Gaio, Ben Perks, Leah S. Hohman, Bruna Araújo David, Paul Kubes, Matthias Mack, Ehud Inbar, Nathan C. Peters

The paper shows that pre-existing Th1 immunity is overcome by monocyte-derived macrophages that create a pathogen niche, and that checkpoint blockade or depletion of these cells improves parasite control.

bioRxiv · Aug 19, 2026 · PreprintImmunology