Author

Christina Müller

Johannes Gutenberg University Mainz

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Eva‐Maria Krämer‐AlbersiD· 2 shared Hauke B. WerneriD· 2 shared Klaus‐Armin NaveiD· 2 shared Wiebke MöbiusiD· 2 shared Anna M. SteyeriD· 1 shared Carsten Frühbeis· 1 shared Dominik FröhlichiD· 1 shared Kerstin Barth· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersNeuroscience

Eva‐Maria Krämer‐AlbersORCID

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2 papers

Recent·Most cited

Progressive axonopathy when oligodendrocytes lack the myelin protein CMTM5

Tobias J. Buscham, Maria A. Eichel‐Vogel, Anna M. Steyer, Olaf Jahn, Nicola Strenzke, Rakshit Dardawal, Tor Rasmus Memhave, Sophie B. Siems, Christina Müller, Martin Meschkat, Ting Sun, Torben Ruhwedel, Wiebke Möbius, Eva‐Maria Krämer‐Albers, Susann Boretius, , Hauke B. Werner

This study shows that CMTM5 deficiency in oligodendrocytes leads to progressive axonopathy, suggesting its role in maintaining axonal integrity rather than myelin formation.

eLife · Feb 28, 2022Peer-reviewedNeuroscience

Oligodendrocytes support axonal transport and maintenance via exosome secretion

Carsten Frühbeis, Wen Ping Kuo, Christina Müller, Kerstin Barth, Leticia Peris, Stefan Tenzer, Wiebke Möbius, Hauke B. Werner, Klaus‐Armin Nave, Dominik Fröhlich, Eva‐Maria Krämer‐Albers

This study shows that oligodendrocyte exosome secretion supports axonal transport and neuronal viability, and its impairment leads to axonal degeneration in mouse mutants.

PLoS Biology · Dec 22, 2020Peer-reviewedNeuroscience