Author

Tobias J. Buscham

Max Planck Institute for Multidisciplinary SciencesORCID 0000-0002-6916-3102Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Neuroscience· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:mouse· 2 Organism:zebrafish· 1

Frequent Co-Authors

Anna M. SteyeriD· 2 shared Hauke B. WerneriD· 2 shared Maria A. Eichel‐VogeliD· 2 shared Wiebke MöbiusiD· 2 shared Ben EmeryiD· 1 shared Christina Müller· 1 shared David Anthony LyonsiD· 1 shared Eva‐Maria Krämer‐AlbersiD· 1 shared

Activity by Year

2022-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersNeuroscience

Anna M. SteyerORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Developmental axon diameter growth of central nervous system axons does not depend on ensheathment or myelination by oligodendrocytes

Jenea M. Bin, Katie Emberley, Tobias J. Buscham, Maria A. Eichel‐Vogel, Ryan A. Doan, Anna M. Steyer, Matthew F. Nolan, Wiebke Möbius, Kelly R. Monk, Hauke B. Werner, Ben Emery, David Anthony Lyons

This study shows that CNS axon diameter growth does not require oligodendrocyte ensheathment or myelination, contrasting with PNS findings.

bioRxiv · Jan 10, 2025 · PreprintNeuroscience

Progressive axonopathy when oligodendrocytes lack the myelin protein CMTM5

Tobias J. Buscham, Maria A. Eichel‐Vogel, Anna M. Steyer, Olaf Jahn, Nicola Strenzke, Rakshit Dardawal, Tor Rasmus Memhave, Sophie B. Siems, Christina Müller, Martin Meschkat, Ting Sun, Torben Ruhwedel, Wiebke Möbius, Eva‐Maria Krämer‐Albers, Susann Boretius, , Hauke B. Werner

This study shows that CMTM5 deficiency in oligodendrocytes leads to progressive axonopathy, suggesting its role in maintaining axonal integrity rather than myelin formation.

eLife · Feb 28, 2022Peer-reviewedNeuroscience