Author

Christine Henes

University of Tübingen

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Blanca Arango‐GonzáleziD· 2 shared Bowen CaoiD· 2 shared Ellen KilgeriD· 2 shared Marius UeffingiD· 2 shared Mathias W. SeeligeriD· 2 shared Regine L. MühlfriedeliD· 2 shared Sylvia Bolz· 2 shared Alexander GünteriD· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 43 mutual co-author links

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2 shared papersNeuroscience

Blanca Arango‐GonzálezORCID

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2 papers

Recent·Most cited

Ocular delivery of different VCP inhibitory formulations prevents retinal degeneration in rhodopsin 255 isoleucine deletion mice

Bowen Cao, Regine L. Mühlfriedel, Merve Sen, Ana-Cristina Almansa-Garcia, Mathias W. Seeliger, Anne-Sophie Petremann-Dumé, Ellen Kilger, Anneli Vollert, Sylvia Bolz, Christine Henes, Paolo Caliceti, Stefano Salmaso, Marius Ueffing, Blanca Arango‐González

This study evaluates three VCP-targeted therapies in a mouse model of retinitis pigmentosa, finding that nanocarrier-encapsulated ML240 provides the best protection against photoreceptor degeneration.

bioRxiv · Jun 25, 2025 · PreprintNeuroscience

Autosomal dominant Retinitis Pigmentosa caused by the rhodopsin isoleucine 255 deletion features rapid neuroretinal degeneration, decreased synaptic connectivity, and neuroinflammation.

Bowen Cao, Yu Zhu, Alexander Günter, Ellen Kilger, Sylvia Bolz, Christine Henes, Regine Mühlfriedel, Mathias W. Seeliger, François Paquet‐Durand, Blanca Arango‐González, Marius Ueffing

This paper introduces a novel mouse model of dominant retinitis pigmentosa caused by the RHO{Delta}I255 mutation, showing rapid photoreceptor degeneration, neuroinflammation, and disrupted neuronal connectivity.

bioRxiv · Aug 29, 2024 · PreprintPathology