Author

D. Chandler-Brown

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Research Focus:chromatin & epigenetic regulation· 2 Research Focus:development & morphogenesis· 2 Approach:experimental assay· 2 Approach:deep learning· 1 Assay:DNA methylation· 1 Research Focus:drug response / drug discovery· 1 Assay:long-read sequencing· 1

Frequent Co-Authors

A. E. Ikui· 1 shared A. Kundaje· 1 shared A. M. Bueno· 1 shared A. Rudner· 1 shared D. S. Tsao· 1 shared G. Marinov· 1 shared J. Kim· 1 shared J. M. Skotheim· 1 shared

Activity by Year

2019-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperCell Biology

A. E. Ikui

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Size-independent mRNA synthesis and chromatin-based partitioning mechanisms generate and maintain constant amounts of protein per cell

M. P. Swaffer, J. Kim, D. Chandler-Brown, M. Langhinrichs, G. Marinov, W. Greenleaf, A. Kundaje, K. M. Schmoller, J. M. Skotheim

The paper identifies transcriptional and chromatin-based mechanisms that generate and maintain constant protein amounts per cell despite changes in cell size.

bioRxiv · Aug 28, 2020Cell Biology

A Highly Scalable and Rapidly Deployable RNA Extraction-Free COVID-19 Assay by Quantitative Sanger Sequencing

D. Chandler-Brown, A. M. Bueno, O. Atay, D. S. Tsao

This paper introduces a Sanger sequencing-based COVID-19 assay that skips RNA extraction, achieving detection limits comparable to qPCR and enabling high-throughput testing.

bioRxiv · Apr 7, 2020 · PreprintMicrobiology

PP2ACdc55 dephosphorylates Pds1 to inhibit spindle elongation

S. Khondker, S. Kajjo, D. Chandler-Brown, J. Skotheim, A. Rudner, A. E. Ikui

The paper shows PP2ACdc55 dephosphorylates Pds1, disrupting its interaction with Esp1 to inhibit spindle elongation during replication stress.

bioRxiv · May 29, 2019 · PreprintCell Biology