Author

D. F. Mosher

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Organism:bacteria (other)· 1 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

D. S. Annis· 2 shared A. S. Hebert· 1 shared C. S. Pepperell· 1 shared D. P. Reinhardt· 1 shared F. J. Fogerty· 1 shared H. N. Poinar· 1 shared J. J. Coon· 1 shared J. M. Mitchell· 1 shared

Activity by Year

2017-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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2 shared papersCell Biology

D. S. Annis

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Author profile →

3 papers

Recent·Most cited

Mutations in Fibronectin Dysregulate Chondrogenesis in Skeletal Dysplasia

N. E. H. Dinesh, J. Rousseau, D. F. Mosher, M. Strauss, J. Mui, P. M. Campeau, D. P. Reinhardt

This paper investigates how fibronectin mutations impair chondrogenesis in skeletal dysplasia by disrupting protein secretion and causing cellular stress in mesenchymal stem cells.

bioRxiv · Dec 22, 2023Cell Biology

Molecular anatomy of eosinophil activation by IL5 and IL33

J. M. Mitchell, J. W. Mabin, L. K. Muehlbauer, D. S. Annis, S. K. Mathur, M. W. Johansson, A. S. Hebert, F. J. Fogerty, J. J. Coon, D. F. Mosher

This study identifies common and distinct phosphoproteomic effects of IL5 and IL33 on human eosinophils, revealing pathways involved in cytoskeletal reorganization, adhesion, and transcriptional regulation.

bioRxiv · Dec 21, 2022 · PreprintCell Biology

Adaptation in a fibronectin binding autolysin of Staphylococcus saprophyticus

T. D. Mortimer, D. S. Annis, M. B. O'Neill, L. L. Bohr, T. M. Smith, H. N. Poinar, D. F. Mosher, C. S. Pepperell

The paper identifies a mutation in the aas gene of Staphylococcus saprophyticus that may enhance its ability to colonize the urinary tract, contributing to its pathogenicity.

bioRxiv · Nov 10, 2017Microbiology