Author

D. J. Kass

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 3 Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:human· 3 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:metagenomics· 1 Organism:mouse· 1

Frequent Co-Authors

J. F. McDyer· 2 shared J. K. Alder· 2 shared N. Kaminski· 2 shared Y. Zhang· 2 shared A. Das· 1 shared A. Gleiberman· 1 shared A. H. Wells· 1 shared A. Morris· 1 shared

Activity by Year

2021-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersCell Biology

J. F. McDyer

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Toll-Like-Receptor 5 protects against pulmonary fibrosis by reducing lung dysbiosis

A. Gleiberman, R. Invernizzi, P. L. Molyneaux, B. G. Wu, Y. Lipinski, M. Kugler, L. N. Segal, Y. Sakamachi, E. Wiley, A. Solis, C. G. Johnson, X. Meng, J. Malphurs, B. Papas, S. Mehta, , N. Kaminski

TLR5 deficiency is linked to pulmonary fibrosis due to its role in preventing microbial dysbiosis following lung injury.

bioRxiv · Apr 30, 2024 · PreprintImmunology

High CXCL6 drives matrix expression and correlate with markers of poor outcome in IPF

H. Bahudhanapati, J. Tan, R.-M. Apel, B. Seeliger, X. Li, T.-Y. Chen, D. Sullivan, J. Sembrat, M. Rojas, E. Valenzi, R. A. Lafyatis, C. Jawale, P. Biswas, J. Tedrow, T. S. Adams, , D. J. Kass

This study identifies CXCL6 as a pro-fibrotic factor in IPF, showing it reduces cAMP, increases collagen and -SMA, and correlates with poor patient survival.

bioRxiv · Jun 22, 2021 · PreprintCell Biology

CD4+ T cell lymphopenia and dysfunction in severe COVID-19 disease is autocrine TNF-α/TNFRI-dependent

I. Popescu, M. E. Snyder, C. J. Iasella, S. J. Hannan, R. Koshy, R. Burke, A. Das, M. J. Brown, E. J. Lyons, S. C. Lieber, X. Chen, J. C. Sembrat, X. An, K. Linstrum, G. Kitsios, , J. F. McDyer

The paper shows that autocrine TNF-α/TNFRI signaling drives CD4+ T cell lymphopenia and dysfunction in severe COVID-19, and TNF-α blockade improves outcomes.

bioRxiv · Jun 2, 2021 · PreprintImmunology