Author

D. Langlais

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 3

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Organism:human· 3 Research Focus:differential expression· 2 Assay:scRNA-seq· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1 Assay:ChIP-seq· 1

Frequent Co-Authors

A. Bousfiha· 1 shared A. J. Martins· 1 shared A. Puel· 1 shared A. Sood· 1 shared C. A. Croft· 1 shared C. A. Piccirillo· 1 shared C. Gruber· 1 shared C. Ma· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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1 shared paperImmunology

A. Bousfiha

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Author profile →

3 papers

Recent·Most cited

The extrafollicular B cell response is a hallmark of childhood idiopathic nephrotic syndrome.

T.-A. Al-Aubodah, L. Aoudjit, G. Pascale, M. Perinpanayagam, D. Langlais, M. Bitzan, S. M. Samuel, C. A. Piccirillo, T. Takano

The paper identifies an extrafollicular B cell response as a key feature of active childhood idiopathic nephrotic syndrome using single-cell RNA-sequencing and flow cytometry.

bioRxiv · Sep 11, 2023Immunology

Pre-existing chromatin accessibility and gene expression differences among naïve CD4+ T cells influence effector potential

D. Rogers, A. Sood, H. Wang, J. J. P. van Beek, T. J. Rademaker, P. Artusa, C. Schneider, C. Shen, D. C. Wong, M.-E. Lebel, S. A. Condotta, M. J. Richer, A. J. Martins, J. S. Tsang, L. Barreiro, , J. N. Mandl

The paper shows pre-existing transcriptional and chromatin differences in naive CD4+ T cells influence their effector lineage potential.

bioRxiv · Apr 21, 2021Immunology

Human T-bet governs innate and innate-like adaptive IFN-γ immunity against mycobacteria

R. Yang, F. Mele, L. Worley, D. Langlais, J. Rosain, I. Benhsaien, H. Elarabi, C. A. Croft, J.-M. Doisne, P. Zhang, M. Weisshaar, D. Jarrossay, D. Latorre, Y. Shen, J. Han, , L. H Glimcher

This study identifies T-bet deficiency as a cause of mycobacterial disease by impairing IFN-γ production in innate and innate-like adaptive lymphocytes.

bioRxiv · Aug 31, 2020Immunology