Author

D. R. Calabrese

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Biochemistry· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1 Assay:CITE-seq· 1

Frequent Co-Authors

A. Shemesh· 2 shared J. R. Greenland· 2 shared L. L. Lanier· 2 shared A. Ferre-D'Amare· 1 shared D. G. Chen· 1 shared D. Incarnato· 1 shared J. Arakawa-Hoyt· 1 shared J. R. Heath· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersImmunology

A. Shemesh

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

CITE-seq analysis reveals human cytomegalovirus and diabetes-associated adaptive NK cell alterations in cardiovascular disease.

S. S. Armstrong, D. G. Chen, S. Kumar, J. R. Heath, M. Feinstein, J. R. Greenland, D. R. Calabrese, L. L. Lanier, K. Ley, A. Shemesh

The paper uses CITE-seq to analyze NK cell alterations in CAD patients, finding SPON2/IFNG ratio and adaptive NK cell signatures correlate with disease severity and inflammation.

bioRxiv · Mar 22, 2024 · PreprintImmunology

Investigating the NRAS 5' UTR as a Target for Small Molecules

S. Balaratnam, Z. R. Torrey, D. R. Calabrese, M. T. Banco, K. Yazdani, X. Liang, A. Ferre-D'Amare, D. Incarnato, J. S. Schneekloth

This study identifies a novel small molecule that binds to the NRAS mRNA G4 structure, showing in vitro translation inhibition but limited in vivo effects, highlighting transcript heterogeneity challenges.

bioRxiv · Jan 5, 2022Biochemistry

Natural Killer cell division regulates FcεRIγ expression downstream of mTOR activity

A. Shemesh, D. R. Calabrese, J. Arakawa-Hoyt, J. R. Greenland, L. L. Lanier

The paper shows that NK cell division and mTOR activity regulate FcRγ expression, with rapamycin and TGFβ reducing FcRγ levels by suppressing division.

bioRxiv · Aug 3, 2021Immunology