Author

D. T. Sessions

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cell Biology· 1

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Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Approach:experimental assay· 1 Approach:mechanistic / dynamical model· 1 Assay:metabolomics· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

D. F. Kashatus· 1 shared H. Sesaki· 1 shared J. A. Kashatus· 1 shared K.-B. Kim· 1 shared K.-S. Park· 1 shared M. W. Mayo· 1 shared N. Churchill· 1 shared

Activity by Year

2022-2022 · peak 1/yr

Co-Author Collaboration Network

7 top collaborators · 21 mutual co-author links

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1 shared paperCell Biology

D. F. Kashatus

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1 paper

Recent·Most cited

Opa1 and Drp1 reciprocally regulate cristae morphology, ETC function, and NAD+ regeneration in KRas-mutant lung adenocarcinoma

D. T. Sessions, K.-B. Kim, J. A. Kashatus, N. Churchill, K.-S. Park, M. W. Mayo, H. Sesaki, D. F. Kashatus

This study shows that Opa1, not Drp1, is critical for maintaining ETC function and NAD+ regeneration in KRas-mutant lung adenocarcinoma, with Opa1 deletion inhibiting tumor growth.

bioRxiv · Jun 1, 2022Cell Biology