Author

D. F. Kashatus

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1 Approach:mechanistic / dynamical model· 1

Frequent Co-Authors

A. C. Dudley· 1 shared A. Rohani· 1 shared C. P. Riedstra· 1 shared D. T. Sessions· 1 shared H. Sesaki· 1 shared J. A. Kashatus· 1 shared K.-B. Kim· 1 shared K.-S. Park· 1 shared

Activity by Year

2022-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCancer Biology

A. C. Dudley

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Melanoma cells appropriate pericyte:endothelial cell crosstalk during perivascular invasion in the brain

Y. Zhang, C. P. Riedstra, S. Wang, S. Patel, T. Bald, P. Kumar, A. Rohani, D. F. Kashatus, S. J. Moschos, A. C. Dudley

This study identifies how melanoma cells use pericyte:endothelial cell signaling during brain invasion, showing that Sox9hi mesenchymal cells use Snail1-dependent mechanisms while Sox10hi cells form clusters.

bioRxiv · Jul 15, 2022 · PreprintCancer Biology

Opa1 and Drp1 reciprocally regulate cristae morphology, ETC function, and NAD+ regeneration in KRas-mutant lung adenocarcinoma

D. T. Sessions, K.-B. Kim, J. A. Kashatus, N. Churchill, K.-S. Park, M. W. Mayo, H. Sesaki, D. F. Kashatus

This study shows that Opa1, not Drp1, is critical for maintaining ETC function and NAD+ regeneration in KRas-mutant lung adenocarcinoma, with Opa1 deletion inhibiting tumor growth.

bioRxiv · Jun 1, 2022Cell Biology