Author

D. W. Hammers

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Pharmacology & Toxicology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:mouse· 4 Research Focus:development & morphogenesis· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1

Frequent Co-Authors

C. C. Hart· 3 shared Y. i. Lee· 3 shared L. Sweeney· 2 shared E. G. Heimsath· 1 shared G. Gao· 1 shared H. L. Sweeney· 1 shared J. A. Hammer· 1 shared J. Xie· 1 shared

Activity by Year

2021-2022 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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Curved chords = mutual co-authorship

3 shared papersPharmacology & Toxicology

C. C. Hart

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Potential limitations of micro-dystrophin gene therapy for Duchenne muscular dystrophy

C. C. Hart, Y. i. Lee, J. Xie, G. Gao, D. W. Hammers, L. Sweeney

This study evaluates micro-dystrophin gene therapy in a DMD mouse model, finding cardiac disease acceleration with one micro-dystrophin and potential benefits with another, highlighting the need for careful dosing.

bioRxiv · Oct 2, 2022Pharmacology & Toxicology

Evaluation of the DBA/2J mouse as a potential background strain for genetic models of cardiomyopathy

C. C. Hart, Y. i. Lee, D. W. Hammers, H. L. Sweeney

The study evaluates the DBA/2J mouse as a background strain for genetic models of cardiomyopathy and finds no evidence of hypertrophic cardiomyopathy or other cardiac pathologies.

bioRxiv · May 16, 2022Physiology

NOX4 inhibition promotes the remodeling of dystrophic muscle

D. W. Hammers

This study shows that inhibiting NOX4 reduces fibrosis and promotes muscle regeneration in dystrophic models of Duchenne muscular dystrophy by targeting myofibroblasts and restoring satellite cells.

bioRxiv · Jan 8, 2022Cell Biology

Filopodia powered by class X myosin promote fusion of mammalian myoblasts

D. W. Hammers, C. C. Hart, M. K. Matheny, E. G. Heimsath, Y. i. Lee, J. A. Hammer, R. E. Cheney, L. Sweeney

This study identifies class X myosin (Myo10) as a driver of filopodia formation that promotes myoblast fusion in muscle development and regeneration.

bioRxiv · Jul 28, 2021Cell Biology