Author

E. Abu-El-Rub

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell-cell communication· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

G. M. Hatch· 3 shared H. M. Zegallai· 3 shared A. J. Marshall· 2 shared L. K. Cole· 2 shared E. M. Mejia· 1 shared F. Olayinka-Adefemi· 1 shared G. C. Sparagna· 1 shared J. Field· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

9 top collaborators · 30 mutual co-author links

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3 shared papersImmunology

G. M. Hatch

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3 papers

Recent·Most cited

Tafazzin knockdown in murine mesenchymal stem cells enhances the tafazzin knockdown mediated elevation in interleukin-10 secretion from murine B lymphocytes

H. M. Zegallai, E. Abu-El-Rub, G. M. Hatch

The paper shows that Taz deficiency in murine MSCs enhances IL-10 secretion from Taz-deficient B cells when co-cultured.

bioRxiv · Mar 1, 2023Immunology

Tafazzin deficiency in mouse mesenchymal stem cells potentiates their immunosuppression and impairs activated B lymphocyte immune function

H. M. Zegallai, E. Abu-El-Rub, F. Olayinka-Adefemi, L. K. Cole, G. C. Sparagna, A. J. Marshall, G. M. Hatch

This study shows that Tafazzin deficiency in mouse mesenchymal stem cells enhances their immunosuppressive effects on activated B lymphocytes.

bioRxiv · Sep 7, 2021Immunology

Tafazzin regulates the function of lipopolysaccharide activated B lymphocytes in mice

H. M. Zegallai, E. Abu-El-Rub, L. K. Cole, J. Field, E. M. Mejia, J. W. Gordon, A. J. Marshall, G. M. Hatch

This study shows that tafazzin deficiency impairs B cell function and metabolism in response to lipopolysaccharide in mice.

bioRxiv · May 17, 2021Immunology