Author

E. Feraille

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

A. Chassot· 2 shared A. SASSI· 2 shared V. Olivier· 2 shared A. Doucet· 1 shared A. Faivre· 1 shared A. Hertig· 1 shared A. P. McMahon· 1 shared C. A. Wagner· 1 shared

Activity by Year

2019-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersPhysiology

A. Chassot

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Aldosterone mediates potassium-induced kidney fibrosis through tubular and mesenchymal mineralocorticoid receptor activation

T. CHARLEMAGNE, A. SASSI, M. Gjorgjieva, r. yvon, M. Tournier, M. Fernandez, A. Chassot, I. Roth, C. Veyrat-Durebex, S. Ramakrishnan, J. Rutkowski, E. Feraille, V. Olivier

This study shows high dietary potassium worsens kidney fibrosis in mice by activating MR in tubular and mesenchymal cells, mediated by aldosterone.

bioRxiv · Jun 15, 2022 · PreprintPhysiology

Claudin-4, a core component of the tight-junctional complex along the collecting system, is induced in nephrotic syndrome

V. Olivier, A. SASSI, G. Arnoux, R. Chambrey, I. Monnay, A. Chassot, K. Udwan, E. Dizin, J. M. Rutkowski, L. Cheval, G. Crambert, C. A. Wagner, A. Doucet, D. Eladari, S. Moll, , S. K Ramakrishnan

This study shows claudin-4 expression increases in nephrotic syndrome, suggesting a shift from transcellular to paracellular chloride transport in the kidney collecting system.

bioRxiv · Jun 1, 2022 · PreprintPhysiology

Impaired renal gluconeogenesis is a major determinant of acute kidney injury associated mortality

D. Legouis, S.-E. Ricksten, A. Faivre, K. Gariani, P. Galichon, L. Berchtold, E. Feraille, K. Koppitch, A. Hertig, P.-Y. Martin, M. Naesens, J. Pugin, A. P. McMahon, P. E. Cippà, S. de Seigneux

This study shows impaired renal gluconeogenesis during AKI contributes to systemic metabolic disturbances and mortality, with thiamine supplementation reducing mortality in critically ill patients.

bioRxiv · Oct 16, 2019Cell Biology