Author

P. Galichon

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:human· 4 Research Focus:cell signaling & trafficking· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Assay:proteomics· 2 Research Focus:cell type annotation· 1

Frequent Co-Authors

D. Legouis· 2 shared J. Hadchouel· 2 shared J. V. Bonventre· 2 shared L. Li· 2 shared M. T. Valerius· 2 shared S. Vandermeersch· 2 shared A. C. Figueroa-Ramirez· 1 shared A. Corchia· 1 shared

Activity by Year

2019-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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Curved chords = mutual co-authorship

2 shared papersPhysiology

D. Legouis

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Protective role of podocytic IL-15/STAT5 pathway in experimental focal and segmental glomerulosclerosis

A. Niasse, K. Louis, O. Lenoir, C. Schwarz, X. Xu, A. Couturier, H. DOBOSZIEWICZ, A. Corchia, S. Placier, S. Vandermeersch, L. Hennighausen, P. Frere, P. Galichon, B. Surin, S. Ouchelouche, , Y. Luque

This study identifies the podocytic IL-15/STAT5 pathway as a protective mechanism in FSGS, showing that IL-15 alleviates glomerular injury by maintaining autophagy.

bioRxiv · Nov 7, 2022 · PreprintPathology

Orphan nuclear receptor COUP-TFII drives the myofibroblast metabolic shift leading to fibrosis

L. Li, P. Galichon, X. Xiao, A. C. Figueroa-Ramirez, D. Tamayo, J. J. Lee, M. Kalocsay, D. Gonzalez-Sanchez, M. S. Chancay, K. McCracken, D. Lemos, N. Lee, T. Ichimura, Y. Mori, M. T. Valerius, , J. V. Bonventre

This study identifies COUP-TFII as a driver of myofibroblast metabolic shift to aerobic glycolysis, promoting fibrosis in multiple organs.

bioRxiv · Jun 5, 2020 · PreprintCell Biology

Proliferation impairs cell viability via energy depletion

P. Galichon, M. Lannoy, L. Li, S. Vandermeersch, D. Legouis, M. T. Valerius, J. Hadchouel, J. V. Bonventre

The paper shows that proliferation in kidney cells reduces energy availability, leading to cell death and chronic kidney disease under metabolic stress.

bioRxiv · Apr 24, 2020 · PreprintPhysiology

Impaired renal gluconeogenesis is a major determinant of acute kidney injury associated mortality

D. Legouis, S.-E. Ricksten, A. Faivre, K. Gariani, P. Galichon, L. Berchtold, E. Feraille, K. Koppitch, A. Hertig, P.-Y. Martin, M. Naesens, J. Pugin, A. P. McMahon, P. E. Cippà, S. de Seigneux

This study shows impaired renal gluconeogenesis during AKI contributes to systemic metabolic disturbances and mortality, with thiamine supplementation reducing mortality in critically ill patients.

bioRxiv · Oct 16, 2019Cell Biology