Author

E. Hollville

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

M. Deshmukh· 2 shared S. Kapoor· 2 shared A. B. Lopez· 1 shared A. Nakamura· 1 shared C. Flowers· 1 shared C. L. Kurtz· 1 shared C. Lopez-Otin· 1 shared J. Baran-Gale· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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2 shared papersPhysiology

M. Deshmukh

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2 papers

Recent·Most cited

miR-29 is an important driver of aging-related phenotypes

V. Swahari, A. Nakamura, E. Hollville, Y.-H. Hung, M. Kanke, C. L. Kurtz, X. M. Caravia, S. He, J. Krishnamurthy, S. Kapoor, V. Prasad, C. Flowers, M. Beck, J. Baran-Gale, N. Sharpless, , M. Deshmukh

This study identifies miR-29 as a key driver of aging-related phenotypes by showing its upregulation in aging models and its ability to induce aging traits when overexpressed.

bioRxiv · Nov 29, 2022Physiology

Constitutive high expression of NOXA sensitizes human embryonic stem cells for rapid cell death

R. Basundra, S. Kapoor, E. Hollville, N. Kiapour, A. B. Lopez, N. M. Melchiorre, M. Deshmukh

This study shows that high NOXA levels in hES cells antagonize MCL1, enabling rapid apoptosis in response to DNA damage.

bioRxiv · Jan 13, 2021Cell Biology