Author

E. Tolstik

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Approach:experimental assay· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

K. Lorenz· 2 shared A. Klinke· 1 shared B. A. Freeman· 1 shared B. Stratmann· 1 shared C. Welke· 1 shared D. Führer· 1 shared D. Geist· 1 shared D. R. Engel· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersPathology

K. Lorenz

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2 papers

Recent·Most cited

Enhanced cardiac mitochondrial biogenesis by nitro-oleic acid remedies diastolic dysfunction in a mouse model of heart failure with preserved ejection fraction

M. Mueller, T. Schubert, C. Welke, T. J. Schulz, T. Patschkowski, T. Maske, L. A. Lengenfelder, L. Landwehrjohann, E. Donhauser, E. T. Vogt, B. Stratmann, J. Hense, S. Luedtke, M. Duefer, E. Tolstik, , A. Klinke

NO2-OA treatment improves diastolic dysfunction in HFpEF mice by enhancing mitochondrial biogenesis and oxidative phosphorylation.

bioRxiv · Feb 20, 2024 · PreprintPathology

Canonical and noncanonical contribution of thyroid hormone receptor isoforms alpha and beta to cardiac hypertrophy and heart rate in male mice

D. Geist, G. S. Hönes, S. C. Grund, J. Pape, D. Siemes, P. Spangenberg, E. Tolstik, S. Dörr, N. Spielmann, H. Fuchs, V. Gailus-Durner, M. Hrabe de Angelis, J. Mittag, D. R. Engel, D. Führer, , L. C. Moeller

This study shows that thyroid hormone-induced cardiac hypertrophy relies on noncanonical TR action, while heart rate regulation depends on canonical TR action, with TRβ compensating for TR absence.

bioRxiv · Nov 24, 2023Physiology