Author

F. Bridoux

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Approach:experimental assay· 3 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Organism:human· 1 Assay:protein structure· 1

Frequent Co-Authors

A. Jaccard· 3 shared C. Sirac· 3 shared M. V. Ayala· 3 shared S. Bender· 3 shared V. Javaugue· 3 shared A. Bonaud· 2 shared A. Rinsant· 2 shared C. Oblet· 2 shared

Activity by Year

2019-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersPathology

A. Jaccard

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

A mouse model of cardiac AL amyloidosis unveils mechanisms of tissue accumulation and toxicity of amyloid fibrils

G. Martinez-Rivas, M. V. Ayala, S. Bender, G. R. Codo, K. W. Swiderska, A. Lampis, L. Pedroza, M. Merdanovic, P. Sicard, E. Pinault, L. Richard, F. Lavatelli, D. Canetti, A. Rinsant, K. Sihem, , C. Sirac

This paper develops a mouse model of cardiac AL amyloidosis that reproduces human disease features, including amyloid deposits and cardiac dysfunction, and identifies a role for partial LC degradation in fibril formation.

Nature Communications · Mar 27, 2025Peer-reviewedPathology

Full-length immunoglobulin high-throughput sequencing reveals specific novel mutational patterns in POEMS syndrome

S. Bender, V. Javaugue, A. Saintamand, M. V. Ayala, M. Alizadeh, M. Filloux, V. Pascal, N. Gachard, D. Lavergne, F. Auroy, M. Cogne, F. Bridoux, C. Sirac, A. Jaccard

This study uses high-throughput Ig repertoire sequencing to identify specific mutational patterns in POEMS syndrome, aiding in diagnosis and detection of low-abundance plasma cell clones.

bioRxiv · Oct 31, 2019Immunology

Glomerulosclerosis and kidney failure in a mouse model of monoclonal immunoglobulin light-chain deposition disease

S. Bender, M. V. Ayala, A. Bonaud, V. Javaugue, C. Carrion, C. Oblet, A. Rinsant, N. Quellard, S. Kaaki, Z. Oruc, F. Boyer, A. Paquet, N. Pons, B. Herve, M. O. Ashi, , C. Sirac

This study develops a mouse model of light chain deposition disease, showing that pathogenic light chains cause glomerulosclerosis and kidney failure, and identifies potential therapeutic targets.

bioRxiv · May 2, 2019 · PreprintPathology