Author

N. Gachard

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:clustering· 1 Assay:DNA methylation· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. Saintamand· 2 shared J. Feuillard· 2 shared A. Jaccard· 1 shared C. El Hamel· 1 shared C. Ouk· 1 shared C. Sirac· 1 shared C. VINCENT-FABERT· 1 shared D. Lavergne· 1 shared

Activity by Year

2019-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersImmunology

A. Saintamand

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Increased frequencies of IgH locus suicide recombination points on Chronic Lymphocytic Leukemia with low rate of AID related somatic mutations, cMYC overexpression and short telomeres

I. Al Jamal, M. Parquet, H. Boutouil, K. Guiyedi, D. Rizzo, M. Dupont, M. Boulin, C. El Hamel, J. Lerat, S. Aoufouchi, S. Al Hamaoui, N. Makdissy, J. Feuillard, N. Gachard, S. Peron

The paper identifies a subgroup of CLL patients with increased IgH locus suicide recombination (LSR) rates, linked to low AID expression, short telomeres, and high cMYC expression, suggesting an AID-independent mechanism for LSR.

bioRxiv · Mar 6, 2022 · PreprintCancer Biology

Full-length immunoglobulin high-throughput sequencing reveals specific novel mutational patterns in POEMS syndrome

S. Bender, V. Javaugue, A. Saintamand, M. V. Ayala, M. Alizadeh, M. Filloux, V. Pascal, N. Gachard, D. Lavergne, F. Auroy, M. Cogne, F. Bridoux, C. Sirac, A. Jaccard

This study uses high-throughput Ig repertoire sequencing to identify specific mutational patterns in POEMS syndrome, aiding in diagnosis and detection of low-abundance plasma cell clones.

bioRxiv · Oct 31, 2019Immunology

B-cell enforced expression of the mouse ortholog of MYD88L265P is responsible for Waldenstrom-like B-cell lymphoma.

C. Ouk, L. Roland, A. Saintamand, N. Gachard, M. Thomas, M. Deveza, N. Faumont, J. Feuillard, C. VINCENT-FABERT

This study shows that enforced expression of MYD88L252P in B-cells leads to a lymphoplasmocytic B-cell lymphoma with a serum IgM peak in mice.

bioRxiv · Oct 6, 2019 · PreprintCancer Biology