Author

F. Kloprogge

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pharmacology & Toxicology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Organism:bacteria (other)· 1 Research Focus:benchmarking· 1 Approach:deep learning· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Assay:proteomics· 1 Approach:statistical model· 1 Approach:transformer / foundation model· 1

Frequent Co-Authors

T. D. McHugh· 2 shared A. A. Witney· 1 shared A. Maitra· 1 shared A. Sole· 1 shared F. G. Hernandez· 1 shared G. Mundin· 1 shared H. Margaryan· 1 shared J. A. Cordero· 1 shared

Activity by Year

2022-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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2 shared papersMicrobiology

T. D. McHugh

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

The impact of physiological state and environmental stress on bacterial load estimation methodologies for Mycobacterium tuberculosis

A. Maitra, M. Wijk, H. Margaryan, P. Denti, T. D. McHugh, F. Kloprogge

This study evaluates how bacterial growth phase and drug exposure affect bacterial load estimation methods for Mycobacterium tuberculosis.

bioRxiv · May 10, 2024 · PreprintMicrobiology

Named Entity Recognition of Pharmacokinetic parameters in the scientific literature

F. G. Hernandez, Q. Nguyen, V. C. Smith, J. A. Cordero, M. R. Ballester, M. Duran, A. Sole, P. Chotsiri, T. Wattanakul, G. Mundin, W. Lilaonitkul, J. F. Standing, F. Kloprogge

This paper introduces a novel corpus and language models for Named Entity Recognition of pharmacokinetic parameters in scientific literature, achieving high accuracy with BioBERT.

bioRxiv · Feb 12, 2024 · PreprintPharmacology & Toxicology

Understanding emergence of antimycobacterial dose dependent resistance.

F. Kloprogge, J. Ortiz Canseco, L. Phee, Z. Sadouki, K. Kipper, A. A. Witney, N. Stoker, T. D. McHugh

This study investigates how concentration-dependent resistance emerges to isoniazid and rifampicin, showing that genotypic resistance only appears when antibiotic levels fall below MIC, and that isoniazid resistance involves concentration-dependent SNP accumulation.

bioRxiv · Sep 2, 2022 · PreprintPharmacology & Toxicology