Author

G. Hermey

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Assay:proteomics· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:differential expression· 2 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. Kaleem· 1 shared A. M. P. Gjessing· 1 shared A. Manavalan· 1 shared A. Moller· 1 shared A. N. Madsen· 1 shared A. Nykjaer· 1 shared A. S. Rosato· 1 shared B. Holst· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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1 shared paperCell Biology

A. Kaleem

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3 papers

Recent·Most cited

TRPML1 activation ameliorates lysosomal phenotypes in CLN3 deficient retinal pigment epithelial cells

D. Wuenkhaus, R. Tang, K. Nyame, N. N. Laqtom, M. Schweizer, A. S. Rosato, E. K. Kroegsaeter, C. Wollnik, M. Abu-Remaileh, C. Grimm, G. Hermey, R. Kuhn, D. Gruber-Schoffnegger, S. Markmann

This study shows TRPML1 activation reduces lysosomal storage in CLN3-deficient retinal cells but does not restore BMP levels.

bioRxiv · Jun 21, 2023Cell Biology

Soluble SorCS1 binds the insulin receptor to enhance insulin sensitivity.

M. Kjolby, P. Breining, K. M. Pedersen, N. Wellner, K. M. J. Sorensen, A. M. P. Gjessing, D. Januliene, D. M. Vittrup, A. Manavalan, M. Rotman, G. Thotakura, G. Hermey, P. Madsen, C. B. Vaegter, S. Thirup, , A. Nykjaer

This study identifies sol-SorCS1 as a soluble form of SorCS1 that enhances insulin sensitivity by binding the insulin receptor, improving glucose uptake and signaling in myocytes.

bioRxiv · Nov 10, 2020 · PreprintPhysiology

CLN3 regulates endosomal function by modulating Rab7A activity

S. Yasa, G. Modica, E. Sauvageau, A. Kaleem, G. Hermey, S. Lefrancois

This study identifies CLN3's role in regulating Rab7A activity to control endosomal trafficking and autophagy, explaining JNCL pathogenesis.

bioRxiv · May 10, 2019 · PreprintCell Biology