Author

G. P. Takacs

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

J. K. Harrison· 2 shared C. J. Kreiger· 1 shared D. A. Mitchell· 1 shared D. C. Harris· 1 shared D. Luo· 1 shared G. Tian· 1 shared H. G. Anderson· 1 shared J. S. Garcia· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersCancer Biology

J. K. Harrison

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2 papers

Recent·Most cited

Cancer BiologybioRxivMay 15, 2023

Global stability and parameter analysis reinforce therapeutic targets of PD-L1-PD-1 and MDSCs for glioblastoma

H. G. Anderson, G. P. Takacs, D. C. Harris…·T. L. Stepien

This paper develops a tumor-immune model for glioblastoma, identifies key parameters for treatment, and suggests combining immune checkpoint inhibitors with MDSC-targeting therapies.

DOI 10.1007/s00285-023-02027-y

humanstatistical model

𝕏 PostBluesky

Cancer BiologybioRxivJul 8, 2022

Glioma-derived CCL2 and CCL7 mediate migration of immune suppressive CCR2+ myeloid cells into the tumor microenvironment in a redundant manner

G. P. Takacs, C. J. Kreiger, D. Luo…·J. K. Harrison

This study shows that CCL2 and CCL7 from gliomas redundantly recruit CCR2+/CX3CR1+ myeloid cells into the tumor microenvironment, contributing to immune suppression and poor prognosis in GBM.

DOI 10.3389/fimmu.2022.993444

mouseproteomics

𝕏 PostBluesky