Author

G. Rätsch

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Organism:mouse· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

A. Khales· 1 shared Accelerating Medicines Partnership RA/SLE Network· 1 shared C. Rozo· 1 shared D. Kuo· 1 shared D. Rao· 1 shared E. F. DiCarlo· 1 shared F. Zhang· 1 shared G. Schwank· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperCancer Biology

A. Khales

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Author profile →

2 papers

Recent·Most cited

Mutant SF3B1 promotes PDAC malignancy through TGF-β resistance

P. Simmler, T. Mengis, K. Van-Lehmann, A. Khales, T. Thomas, G. Rätsch, M. Stoffel, G. Schwank

This study shows that SF3B1K700E mutation in PDAC enhances tumor aggressiveness when combined with KRAS and p53 mutations by promoting TGF-β resistance.

bioRxiv · Jun 16, 2022Cancer Biology

HBEGF+ macrophages identified in rheumatoid arthritis promote joint tissue invasiveness and are reshaped differentially by medications

D. Kuo, J. Ding, I. Cohn, F. Zhang, K. Wei, D. Rao, C. Rozo, U. K. Sokhi, Accelerating Medicines Partnership RA/SLE Network, E. F. DiCarlo, M. B. Brenner, V. P. Bykerk, S. M. Goodman, S. Raychaudhuri, G. Rätsch, , L. T. Donlin

A newly identified human macrophage phenotype in RA promotes joint tissue invasiveness and shows variable responses to anti-inflammatory medications.

bioRxiv · Jan 31, 2019 · PreprintImmunology