Author

G. Wabnitz

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 3

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 3 Research Focus:cell type annotation· 3 Organism:human· 3 Research Focus:differential expression· 2 Organism:mouse· 2 Approach:statistical model· 2 Research Focus:cell signaling & trafficking· 1 Approach:experimental assay· 1

Frequent Co-Authors

R. Grieshaber-Bouyer· 3 shared C. Mueller-Tidow· 2 shared F. A. Radtke· 2 shared H.-M. Lorenz· 2 shared N. S. Hackert· 2 shared P. A. Nigrovic· 2 shared T. Exner· 2 shared A. Dalpke· 1 shared

Activity by Year

2021-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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3 shared papersImmunology

R. Grieshaber-Bouyer

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3 papers

Recent·Most cited

Human and murine neutrophils share core transcriptional programs in both homeostatic and inflamed contexts

N. S. Hackert, F. A. Radtke, T. Exner, H.-M. Lorenz, C. Mueller-Tidow, P. A. Nigrovic, G. Wabnitz, R. Grieshaber-Bouyer

This study compares transcriptional programs of human and murine neutrophils in homeostasis and inflammation, finding shared core programs despite species differences.

bioRxiv · Nov 13, 2022 · PreprintImmunology

Aging and interferon gamma response drive the phenotype of neutrophils in the inflamed joint

R. Grieshaber-Bouyer, T. Exner, N. S. Hackert, F. A. Radtke, S. A. Jelinsky, O. Halyabar, A. Wactor, E. Karimizadeh, J. Schettini, A. H. Jonsson, D. A. Rao, L. A. Henderson, C. Mueller-Tidow, H.-M. Lorenz, G. Wabnitz, , P. A. Nigrovic

This study identifies transcriptional and phenotypic changes in synovial fluid neutrophils from arthritis patients, highlighting IFNγ response and aging as key drivers.

bioRxiv · Nov 16, 2021 · PreprintImmunology

Inflammation induces pro-NETotic neutrophils via TNFR2 signaling

F. Neuenfeldt, J. C. Schumacher, R. Grieshaber-Bouyer, J. Habicht, J. Schröder-Braunstein, A. Gauss, B. Niesler, N. Heineken, A. Dalpke, M. M. Gaida, T. Giese, S. Meuer, Y. Samstag, G. Wabnitz

The paper identifies CCR5+ cytokine-induced PMN as pro-NETotic neutrophils driven by TNFR2 signaling in chronic inflammation.

bioRxiv · Jun 21, 2021Immunology