Author

J. Habicht

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Immunology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:cell type annotation· 3 Organism:human· 3 Research Focus:cell signaling & trafficking· 2 Assay:flow cytometry· 2 Research Focus:host-pathogen & immune response· 2 Assay:proteomics· 2 Approach:wet-lab / biochemical assay· 2

Frequent Co-Authors

A. Mooneyham· 2 shared F. Neuenfeldt· 2 shared M. Bazzaro· 2 shared M. Shetty· 2 shared N. Heineken· 2 shared R. Grieshaber-Bouyer· 2 shared Y. Samstag· 2 shared A. Dalpke· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersCell Biology

A. Mooneyham

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Author profile →

4 papers

Recent·Most cited

Distinct Functional States of Neutrophils by Actin Disassembly and NF-kB/STAT3 Signaling

G. H. Wabnitz, N. Heineken, J.-C. Schumacher, T. Exner, F. Neuenfeldt, J. Habicht, F. Kahlich, H. Platzer, U. Merle, T. Renkawitz, Y. Samstag, R. Grieshaber-Bouyer, S. Autenrieth

This study identifies two distinct neutrophil states in mice and patients, driven by actin dynamics and NF-kB/STAT3 signaling, with different functional outcomes.

bioRxiv · Jul 14, 2023 · PreprintImmunology

Inflammation induces pro-NETotic neutrophils via TNFR2 signaling

F. Neuenfeldt, J. C. Schumacher, R. Grieshaber-Bouyer, J. Habicht, J. Schröder-Braunstein, A. Gauss, B. Niesler, N. Heineken, A. Dalpke, M. M. Gaida, T. Giese, S. Meuer, Y. Samstag, G. Wabnitz

The paper identifies CCR5+ cytokine-induced PMN as pro-NETotic neutrophils driven by TNFR2 signaling in chronic inflammation.

bioRxiv · Jun 21, 2021Immunology

UNC-45A Weakens and Breaks MT Lattice Independent of its Effect on Non-Muscle Myosin II

M. Bazzaro, J. Habicht, A. Mooneyham, A. Hoshino, M. Shetty, X. Zhang, E. Emmings, Q. Yang, C. Coombes, M. K. Gardner

This study shows UNC-45A weakens and breaks microtubule lattices independently of its effect on non-muscle myosin II, with implications for disease treatment.

bioRxiv · Jun 20, 2020Cell Biology

UNC-45A is preferentially expressed in epithelial cells and binds to and co-localizes with interphase MTs

J. Habicht, A. Mooneyham, M. Shetty, X. Zhang, V. Shridhar, B. Winterhoff, Y. Zhang, J. Cepela, T. Starr, E. Lou, M. Bazzaro

This paper shows UNC-45A is preferentially expressed in epithelial cells and co-localizes with microtubules in interphase cells, independent of actin or myosin.

Cancer Biology & Therapy · Jul 22, 2019Peer-reviewedCell Biology