Author

Gabriela Fort

University of UtahPrimary ORCID profile (0000-0002-9776-4984 · 1 paper)Unclaimed ORCID profile · follow to get notified of new preprints

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Same name, identified by ORCID

Gabriela FortiD1 paper · Huntsman Cancer Institute, University of Utah;

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Eric L. SnyderiD· 2 shared Kayla T. O’TooleiD· 2 shared Adamaris Martinez· 1 shared Anastasia Proveyeka· 1 shared Anna K. Waters· 1 shared Brandon MurphyiD· 1 shared Deevya Baral· 1 shared Elliott L. PaineiD· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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2 shared papersCancer Biology

Eric L. SnyderORCID

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Author profile →

2 papers

Recent·Most cited

Characterization of the BRAF interactome identifies BRAFV600ETP53 interaction in melanoma

Kayla T. O’Toole, Adamaris Martinez, Brandon Murphy, Anastasia Proveyeka, Gabriela Fort, Fatima Al-Sudani, Sanjana Srinivas Boggaram, Elliott L. Paine, Deevya Baral, Joshua Lyon Andersen, Gennie Lynne Parkman, Eric L. Snyder, Robert L. Judson‐Torres, Martin McMahon

The paper identifies a novel interaction between BRAFV600E and TP53 in melanoma, suggesting that BRAFV600E inactivates TP53, explaining the rarity of TP53 alterations in this cancer type.

bioRxiv · Jun 20, 2025 · PreprintCancer Biology

Opposing lineage specifiers induce a pro-tumor hybrid-identity state in lung adenocarcinoma

Gabriela Fort, Henry Arnold, Soledad A. Camolotto, Kayla T. O’Toole, Rushmeen Tariq, Anna K. Waters, Katherine Gillis, Eric L. Snyder

This study identifies HNF4 as a driver of hybrid-identity states in KRAS-driven lung adenocarcinoma, showing that opposing lineage specifiers promote tumor progression and affect therapy response.

bioRxiv · Dec 2, 2024 · PreprintCancer Biology