Author

Caitlin Orr

Abramson Cancer Center

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Genetic & Genomic Medicine· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Kara N. MaxwelliD· 2 shared Ryan M. HausleriD· 2 shared Alexandra IndegliaiD· 1 shared Alexandra O. SokolovaiD· 1 shared Alison Schwartz LevineiD· 1 shared Anne NaumeriD· 1 shared Carolyn S. MenendeziD· 1 shared Catherine Wolfe· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersGenetic & Genomic Medicine

Kara N. MaxwellORCID

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2 papers

Recent·Most cited

Pathogenic germline variants in a racially diverse real-world cohort of prostate cancer patients

Taylor B. Crawford, Maliha N Tayeb, Emanuel Barrett, Tara Al-Saleem, Pranavi Kondam, Ryan M. Hausler, Heather Symecko, Caitlin Orr, Catherine Wolfe, Derek Mann, Kelsey Spielman, Gloria Wong, Naomi Balzer Haas, Vivek Narayan, Kyle William Robinson, , Kara N. Maxwell

This study reports a 5.4% rate of pathogenic germline variants in a racially diverse cohort of prostate cancer patients, with significant associations between PGV rates and age at diagnosis and self-identified race.

Journal of the National Comprehensive Cancer Network · Jun 2, 2026Peer-reviewedGenetic & Genomic Medicine

Variation at the R181 residue of p53 confers loss of p53 DNA binding cooperativity with the retention of mitochondrial-associated apoptosis

Renyta Moses, Alexandra Indeglia, Alison Schwartz Levine, Ryan M. Hausler, Gregory Kelly, Sven A. Miller, Isabel Anez-Bruzual, Melissa Heller, R Delgado, Caitlin Orr, Wendy Kohlmann, Anne Naumer, Jennie Vagher, Sophie R Cahill, Luke Devon Maese, , Kara N. Maxwell

This study shows that p53 variants with R181 mutations retain mitochondrial apoptosis function but lose DNA binding cooperativity, leading to reduced cancer risk.

Molecular Cancer Research · May 18, 2026Peer-reviewedCancer Biology