Author

H. A. Bolck

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:gene regulatory network inference· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1

Frequent Co-Authors

H. Moch· 2 shared A. Bratus-Neuenschwander· 1 shared A. Kahraman· 1 shared A. Vukolic· 1 shared C. Cortijo· 1 shared C. Hirt· 1 shared C. Mueller· 1 shared C. Pauli· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersCancer Biology

H. Moch

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Author profile →

2 papers

Recent·Most cited

Heterogeneous and Novel Transcript Expression in Single Cells of Patient-Derived ccRCC Organoids

T. Karakulak, N. Zajac, H. A. Bolck, A. Bratus-Neuenschwander, Q. Zhang, W. Qi, D. Basu, T. C. Oltra, H. Rehrauer, C. von Mering, H. Moch, A. Kahraman

This study uses single-cell long-read sequencing to analyze transcriptomes of ccRCC organoids, identifying novel transcripts and highlighting cell-specific alternative splicing events.

bioRxiv · Mar 15, 2024 · PreprintCancer Biology

SF3B1 promotes tumor malignancy through splicing-independent co-activation of HIF1α

P. Simmler, C. Cortijo, L. M. Koch, P. Galliker, M. Schaefer, S. Angori, H. A. Bolck, C. Mueller, A. Vukolic, P. Mirtschink, Y. Christinat, N. R. Davidson, K.-V. Lehmann, G. Pellegrini, C. Pauli, , G. Schwank

This study shows SF3B1 promotes tumor malignancy by co-activating HIF1α through a splicing-independent mechanism, enhancing hypoxia response element binding and target gene expression.

bioRxiv · Nov 10, 2020Cell Biology