Author

H. E. Abud

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Approach:statistical model· 1

Frequent Co-Authors

G. Kerr· 2 shared P. J. McMurrick· 2 shared T. Jarde· 2 shared A. De Paoli· 1 shared A. M. Kane· 1 shared A. Mansell· 1 shared B. A. Croker· 1 shared B. A. Leggett· 1 shared

Activity by Year

2019-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersCancer Biology

G. Kerr

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Aspirin synergizes with regorafenib to reduce growth of colorectal cancer

C. Su, L. J. Fennell, C. E. Bond, A. M. Kane, G. Kerr, T. Jarde, D. Micati, R. M. Engel, W. H. Chan, S. Hlavca, S. Archer, P. J. McMurrick, H. Hammerlindl, F. Lim, B. Salik, , V. L. Whitehall

This study shows that aspirin and regorafenib together reduce colorectal cancer growth in preclinical models by inhibiting the PI3K-Akt-mTOR pathway and activating AMPK.

bioRxiv · Feb 11, 2022 · PreprintCancer Biology

SRSF3 confers selective processing of miR-17-92 cluster to promote tumorigenic properties in colorectal cancer

M. Ratnadiwakara, R. Engel, T. Jarde, P. J. McMurrick, H. E. Abud, M.-L. Anko

This study identifies SRSF3's role in selectively processing miR-17-92 cluster miRNAs, promoting tumorigenic properties in colorectal cancer by inhibiting CDKN1A and enhancing cell cycle progression.

bioRxiv · Jun 11, 2019 · PreprintCell Biology

NOD1 mediates non-canonical inflammasome processing of interleukin-18 in epithelial cells to Helicobacter pylori infection.

L. S. Tran, L. Ying, K. D'Costa, G. Wray-McCann, G. Kerr, L. Le, C. C. Allison, J. Ferrand, H. Chaudhry, J. Emery, A. De Paoli, S. Creed, M. Kaparakis-Liaskos, J. Como, J. Dowling, , R. L. Ferrero

This study identifies NOD1 as a key regulator of IL-18 processing in epithelial cells during Helicobacter pylori infection, independent of canonical inflammasome proteins.

bioRxiv · Mar 24, 2019 · PreprintImmunology