Author

A. De Paoli

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Assay:metabolomics· 1 Assay:perturbation screen· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Mansell· 1 shared B. A. Croker· 1 shared B. M. Anderson· 1 shared C. C. Allison· 1 shared C. Giannangelo· 1 shared D. J. Creek· 1 shared D. J. Philpott· 1 shared G. Kerr· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperImmunology

A. Mansell

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2 papers

Recent·Most cited

System-wide biochemical analysis reveals ozonide antimalarials initially act by disrupting Plasmodium falciparum haemoglobin digestion

C. Giannangelo, G. Siddiqui, A. De Paoli, B. M. Anderson, L. E. Edgington-Mitchell, S. A. Charman, D. J. Creek

This paper shows ozonide antimalarials disrupt Plasmodium falciparum haemoglobin digestion, leading to parasite death and stress responses.

PLOS Pathogens · Jun 26, 2020Peer-reviewedMicrobiology

NOD1 mediates non-canonical inflammasome processing of interleukin-18 in epithelial cells to Helicobacter pylori infection.

L. S. Tran, L. Ying, K. D'Costa, G. Wray-McCann, G. Kerr, L. Le, C. C. Allison, J. Ferrand, H. Chaudhry, J. Emery, A. De Paoli, S. Creed, M. Kaparakis-Liaskos, J. Como, J. Dowling, , R. L. Ferrero

This study identifies NOD1 as a key regulator of IL-18 processing in epithelial cells during Helicobacter pylori infection, independent of canonical inflammasome proteins.

bioRxiv · Mar 24, 2019 · PreprintImmunology