Author

Hernan G. Vasquez

Baylor College of Medicine

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:human· 2 Organism:mouse· 2 Assay:spatial transcriptomics· 2 Approach:statistical model· 2 Research Focus:chromatin & epigenetic regulation· 1 Approach:deep learning· 1

Frequent Co-Authors

Ying H. SheniD· 2 shared Abhijit ChakrabortyiD· 1 shared Alan DaughertyiD· 1 shared Anita M DeswaliD· 1 shared Bowen Li· 1 shared Chen ZhangiD· 1 shared Criag Morrell· 1 shared Deborah C. VelaiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersImmunology

Ying H. ShenORCID

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2 papers

Recent·Most cited

Epigenetic Programming of Macrophage Phenotypes by STING-IRF3 Drives Inflammation in Ascending Thoracic Aortic Dissection

Bowen Li, Chen Zhang, Samantha Xu, Yanming Li, Deborah C. Vela, Hernan G. Vasquez, Lin Zhang, Abhijit Chakraborty, Hong S. Lu, Joseph S. Coselli, Toru Suzuki, Alan Daugherty, Dianna M. Milewicz, Ziad Mallat, Liwu Li, , Ying H. Shen

This study identifies STING-IRF3-mediated epigenetic programming of macrophages as a driver of inflammation in aortic dissection, showing its role in pro-inflammatory bias and therapeutic potential.

bioRxiv · Jan 25, 2026 · PreprintImmunology

Downregulation of LATS1/2 Drives Endothelial Senescence-Associated Stemness (SAS) and Atherothrombotic Lesion Formation

Sivareddy Kotla, Jonghae Lee, Kyung Ae Ko, Weiqing Chen, Venkata Subrahman K Samanthapudi, Oanh N. Hoang, Gilbert F. Mejia, Shengyu Li, Keri Schadler, Luis Antonio Rivera, Masaki Imanishi, Kay Carlene Tavares Samperio, Jung Hyun Kim, Keila Carolina Ostos Mendoza, Karla N. Mariscal-Reyes, , Jun‐ichi Abe

This study shows that loss of LATS1/2 in endothelial cells promotes a senescence-associated stemness (SAS) state, leading to atherothrombotic plaque formation through metabolic and mitochondrial dysregulation.

bioRxiv · Jun 19, 2025 · PreprintMolecular Biology