Author

Hong Liang

The University of Texas Health Science Center at Houston

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Same name, identified by ORCID

Biology Disciplines

Click to filter author papers

Cancer Biology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:gene regulatory network inference· 1 Approach:statistical model· 1

Frequent Co-Authors

Alex ReediD· 1 shared Benjamin Cravatt· 1 shared Catherine DenicourtiD· 1 shared Claudia PedrozaiD· 1 shared Han LiaoiD· 1 shared Haoqiang YingiD· 1 shared Jeffrey T. ChangiD· 1 shared Jewon Jung· 1 shared

Activity by Year

2020-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCancer Biology

Alex ReedORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Lipid acyl chain remodeling governs KRAS nanoclustering and oncogenic functions through membrane composition control

Neha Arora, Rafael Bravo, Hong Liang, Jeffrey T. Chang, Alex Reed, Junchen Liu, Benjamin Cravatt, Haoqiang Ying, Claudia Pedroza, Yong Zhou

This study shows that lipid acyl chain remodeling via LPCAT3 affects KRAS nanoclustering and oncogenic activity by altering membrane composition.

bioRxiv · Oct 7, 2026 · PreprintCancer Biology

Elevated MCOLN1 Expression in p53-Deficient Bladder Cancer is Necessary for Oncogene-Induced Cell proliferation, Inflammation, and Invasion

Jewon Jung, Han Liao, Hong Liang, John F. Hancock, Catherine Denicourt, Kartik Venkatachalam

This study shows that loss of p53 in bladder cancer increases MCOLN1 expression, which is necessary for HRAS-driven proliferation, inflammation, and invasion.

bioRxiv · Jul 8, 2020 · PreprintCancer Biology