Author

I. Angelidis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cell Biology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:cell type annotation· 3 Assay:scRNA-seq· 3 Research Focus:cell-cell communication· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1

Frequent Co-Authors

F. J. Theis· 3 shared H. B. Schiller· 3 shared M. Strunz· 3 shared C. H. Mayr· 2 shared M. Ansari· 2 shared A. Chatzigeorgiou· 1 shared A. Cvejic· 1 shared A. Galaras· 1 shared

Activity by Year

2019-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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3 shared papersPharmacology & Toxicology

F. J. Theis

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Cell circuits underlying nanomaterial specific respiratory toxicology

C. Voss, L. Han, M. Ansari, M. Strunz, V. Haefner, C. Ballester-Lopez, I. Angelidis, C. H. Mayr, T. Berthing, T. Conlon, Q. Liu, H. Ren, Q. Zhou, O. Schmid, A. O. Yildirim, , T. Stoeger

This study uses single-cell RNA-sequencing to analyze how different nanomaterials affect lung inflammation in mice, identifying cell type-specific responses and mechanisms.

bioRxiv · Feb 10, 2024 · PreprintPharmacology & Toxicology

Autocrine Sfrp1 inhibits lung fibroblast invasion during transition to injury induced myofibroblasts

C. H. Mayr, A. Sengupta, M. Ansari, J. Pestoni, P. Ogar, I. Angelidis, A. Liontos, J. A. Rodriguez, N. J. Lang, M. Strunz, S. Ashgarpour, D. Gonzalez, M. Gerckens, B. Oehrle, V. Viteri, , H. B. Schiller

The paper identifies Sfrp1 as an autocrine inhibitor of fibroblast invasion during lung fibrosis by analyzing fibroblast states and signaling pathways in mouse lung regeneration.

bioRxiv · Jul 11, 2022Cell Biology

Interferon-induced lysosomal membrane permeabilization and death cause cDC1-deserts in tumors.

E. Aerakis, A. Chatzigeorgiou, M.-M. Alvanou, M. Matthaiakaki-Panagiotaki, I. Angelidis, D. Koumadorakis, A. Galaras, P. Hatzis, D. Kerdidani, M. Makridakis, A. Vlahou, B. Malissen, S. Henri, M. Merad, M. Tsoumakidou

This study identifies interferon-induced lysosomal membrane permeabilization and death as mechanisms causing cDC1 scarcity in tumors, suggesting therapeutic strategies to enhance anti-tumor immunity.

bioRxiv · Mar 14, 2022 · PreprintImmunology

A cellular census of healthy lung and asthmatic airway wall identifies novel cell states in health and disease

F. A. Vieira Braga, G. Kar, M. Berg, O. A. Carpaij, K. Polanski, L. M. Simon, S. Brouwer, T. Gomes, L. Hesse, J. Jiang, E. S. Fasouli, M. Efremova, R. Vento-Tormo, K. Affleck, S. Palit, , S. A. Teichmann

The paper uses single-cell transcriptomics to identify novel cell states in healthy and asthmatic lung tissues, revealing changes in cell communication and immune responses.

bioRxiv · Jan 23, 2019 · PreprintImmunology