Author

A. O. Yildirim

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 2 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Research Focus:cell-cell communication· 2 Research Focus:differential expression· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

C. H. Mayr· 2 shared H. B. Schiller· 2 shared M. Lehmann· 2 shared A. Hilgendorff· 1 shared A. Sengupta· 1 shared B. Ballester Llobell· 1 shared B. Ballester-Llobell· 1 shared C. Ballester-Lopez· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 26 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersPharmacology & Toxicology

C. H. Mayr

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Cell circuits underlying nanomaterial specific respiratory toxicology

C. Voss, L. Han, M. Ansari, M. Strunz, V. Haefner, C. Ballester-Lopez, I. Angelidis, C. H. Mayr, T. Berthing, T. Conlon, Q. Liu, H. Ren, Q. Zhou, O. Schmid, A. O. Yildirim, , T. Stoeger

This study uses single-cell RNA-sequencing to analyze how different nanomaterials affect lung inflammation in mice, identifying cell type-specific responses and mechanisms.

bioRxiv · Feb 10, 2024 · PreprintPharmacology & Toxicology

Primary human lung fibroblasts exhibit trigger- but not disease-specific cellular senescence and impair alveolar epithelial cell progenitor function

N. Bramey, M. C. Melo-Narvaez, F. See, B. Ballester-Llobell, C. Steinchen, E. Jain, K. Hafner, A. O. Yildirim, M. Koenigshoff, M. Lehmann

This study shows that primary human lung fibroblasts from different disease origins exhibit similar susceptibility to senescence induction but distinct senescent programs, which impair alveolar epithelial progenitor function.

bioRxiv · Jul 24, 2023 · PreprintCell Biology

Fibroblasts-derived extracellular vesicles contain SFRP1 and mediate pulmonary fibrosis

O. Burgy, C. H. Mayr, B. Ballester Llobell, A. Sengupta, D. Schenesse, C. Coughlan, T. Parimon, P. Chen, M. Lindner, A. Hilgendorff, M. Mann, A. O. Yildirim, O. Eickelberg, M. Lehmann, H. B. Schiller, , M. Königshoff

This study identifies fibroblast-derived extracellular vesicles containing SFRP1 that contribute to pulmonary fibrosis and suggests SFRP1 as a therapeutic target.

bioRxiv · Dec 22, 2022Cell Biology