Author

J. E. La Marca

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Organism:Drosophila· 2 Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

H. E. Richardson· 2 shared A. Veraksa· 1 shared G. L. Kelly· 1 shared L. M. Parsons· 1 shared M. Portela· 1 shared P. Burke· 1 shared P. O. Humbert· 1 shared R. W. Ely· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersCell Biology

H. E. Richardson

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Author profile →

2 papers

Recent·Most cited

The Drosophila Tumour Suppressor Lgl and Vap33 activate the Hippo pathway by a dual mechanism, involving RtGEF/Git/Arf79F and inhibition of the V-ATPase.

H. E. Richardson, M. Portela, S. Mukherjee, S. Paul, J. E. La Marca, L. M. Parsons, A. Veraksa

This study shows Lgl activates the Hippo pathway via Vap33 through two mechanisms: interacting with RtGEF/Git/Arf79F and inhibiting V-ATPase activity.

bioRxiv · Jul 10, 2023Cell Biology

A Drosophila in vivo chemical screen reveals that combination drug treatment targeting MEK and DGKα mitigates Ras-driven polarity-impaired tumourigenesis.

H. E. Richardson, J. E. La Marca, R. W. Ely, S. T. Diepstraten, P. Burke, G. L. Kelly, P. O. Humbert

This paper identifies a combination of MEK and DGKα inhibitors that reduces Ras-driven tumourigenesis in Drosophila and human cells.

bioRxiv · Mar 14, 2022 · PreprintCancer Biology