Author

J. J. Boyle

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Assay:bulk RNA-seq· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:imaging· 1 Organism:mouse· 1

Frequent Co-Authors

A. Tsao· 1 shared D. Chiappo· 1 shared D. O. J. Haskard· 1 shared E. Forte· 1 shared F. Chugtai· 1 shared F. S. Ng· 1 shared J. Branca· 1 shared J. C. Mason· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperPathology

A. Tsao

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Author profile →

2 papers

Recent·Most cited

Hemin-driven chromatin remodelling by atherosclerotic risk gene SMARCA4 switches human blood-derived macrophages from leukocyte disposal to erythrocyte disposal

L. Cave, K. M. Lodge, D. Chiappo, S. Sinha, F. Chugtai, A. Tsao, J. C. Mason, D. O. J. Haskard, S. E. Humphries, J. J. Boyle

This study shows SMARCA4 drives hemin-induced chromatin changes in macrophages, switching their phagocytic target from leukocytes to erythrocytes, contributing to atherosclerosis.

bioRxiv · May 1, 2023 · PreprintPathology

Myocardial damage induced by a single high dose of isoproterenol in C57BL/6J mice triggers a persistent adaptive immune response against the heart.

E. Forte, M. Panahi, F. S. Ng, J. J. Boyle, J. Branca, O. Bedard, M. G. Hasham, L. Benson, S. E. Harding, N. Rosenthal, S. Sattler

This study shows that a single high dose of isoproterenol in mice causes myocardial damage and a persistent adaptive immune response against the heart, making it a suitable model for studying anti-heart autoimmunity.

bioRxiv · Feb 27, 2020Immunology