Author

F. S. Ng

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Assay:imaging· 2 Approach:deep learning· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

A. Ciacci· 1 shared A. Sau· 1 shared B. Thomas· 1 shared D. J. Hickey· 1 shared E. Forte· 1 shared J. A. Coleman· 1 shared J. Branca· 1 shared J. J. Boyle· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperPhysiology

A. Ciacci

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2 papers

Recent·Most cited

Identifying locations susceptible to micro-anatomical reentry using a spatial network representation of atrial fibre maps

M. Falkenberg, J. A. Coleman, S. Dobson, D. J. Hickey, L. Terrill, A. Ciacci, B. Thomas, N. S. Peters, A. Sau, F. S. Ng, J. Zhao, K. Christensen

This paper introduces a computational method to identify atrial regions susceptible to micro-reentry by modeling percolation-like phenomena on spatial networks of atrial fibre maps.

PLOS ONE · Jun 23, 2022Peer-reviewedPhysiology

Myocardial damage induced by a single high dose of isoproterenol in C57BL/6J mice triggers a persistent adaptive immune response against the heart.

E. Forte, M. Panahi, F. S. Ng, J. J. Boyle, J. Branca, O. Bedard, M. G. Hasham, L. Benson, S. E. Harding, N. Rosenthal, S. Sattler

This study shows that a single high dose of isoproterenol in mice causes myocardial damage and a persistent adaptive immune response against the heart, making it a suitable model for studying anti-heart autoimmunity.

bioRxiv · Feb 27, 2020Immunology