Author

O. Bedard

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Assay:bulk RNA-seq· 1 Assay:imaging· 1 Assay:proteomics· 1

Frequent Co-Authors

N. Rosenthal· 3 shared C. Baker· 2 shared E. Forte· 2 shared A. Chiramel· 1 shared A. Shamsaddini· 1 shared C. Brunton· 1 shared C. Clancy· 1 shared C. M. Lutz· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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3 shared papersImmunology

N. Rosenthal

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Characterization of Collaborative Cross mouse founder strain CAST/EiJ as a novel model for lethal COVID-19

C. Baker, D. Duso, N. Kothapalli, T. Hart, S. Casey, L. Kummer, T. Cookenham, J. Hvizdos, K. Lanzer, P. Vats, P. Shanbhag, I. Bell, M. Tighe, K. Travis, F. Szaba, , N. Rosenthal

The paper characterizes the CAST/EiJ mouse strain as a model for severe COVID-19 by showing it develops high viral burdens, severe lung pathology, and cytokine dysregulation leading to mortality.

bioRxiv · Apr 26, 2024Immunology

Genetically diverse mouse models of SARS-CoV-2 infection reproduce clinical variation in type I interferon and cytokine responses in COVID-19

S. Robertson, O. Bedard, K. McNally, C. Shaia, C. Clancy, M. Lewis, R. Broeckel, A. Chiramel, J. G. Shannon, G. Sturdevant, R. Rosenke, S. L. Anzick, E. Forte, C. Preuss, C. Baker, , S. Best

The paper uses genetically diverse mice to model varied innate immune responses to SARS-CoV-2 infection, showing differences in survival, viral replication, and cytokine profiles.

bioRxiv · Sep 17, 2021Microbiology

Myocardial damage induced by a single high dose of isoproterenol in C57BL/6J mice triggers a persistent adaptive immune response against the heart.

E. Forte, M. Panahi, F. S. Ng, J. J. Boyle, J. Branca, O. Bedard, M. G. Hasham, L. Benson, S. E. Harding, N. Rosenthal, S. Sattler

This study shows that a single high dose of isoproterenol in mice causes myocardial damage and a persistent adaptive immune response against the heart, making it a suitable model for studying anti-heart autoimmunity.

bioRxiv · Feb 27, 2020Immunology