Author

K. A. Wegner

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Organism:mouse· 2 Assay:behavioral assay· 1 Research Focus:host-pathogen & immune response· 1 Assay:imaging· 1

Frequent Co-Authors

C. M. Vezina· 3 shared D. E. Bjorling· 3 shared H. L. Zhang· 2 shared H. M. RUETTEN· 2 shared P. Wang· 2 shared Z. Wang· 2 shared B. Chelgren· 1 shared B. Mueller· 1 shared

Activity by Year

2019-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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3 shared papersPhysiology

C. M. Vezina

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3 papers

Recent·Most cited

A Uropathogenic E. coli UTI89 model of prostatic inflammation and collagen accumulation for use in studying aberrant collagen production in the prostate

H. M. RUETTEN, J. Sandhu, B. Mueller, P. Wang, H. L. Zhang, K. A. Wegner, M. Cadena, S. Sandhu, L. Abler, J. Zhu, C. A. O'Driscoll, B. Chelgren, Z. Wang, T. Shen, J. Barasch, , C. M. Vezina

This paper develops a mouse model of prostatic inflammation and collagen accumulation using E. coli UTI89 instillations to study aberrant collagen production in the prostate.

bioRxiv · Aug 10, 2020Physiology

An immunohistochemical prostate cell identification key indicates that aging shifts procollagen 1A1 production from myofibroblasts to fibroblasts in dogs prone to prostate-related urinary dysfunction

H. Ruetten, C. Cole, M. Wehber, K. A. Wegner, N. M. Girardi, N. T. Peterson, B. R. Scharpf, M. F. Romero, M. W. Wood, S. A. Colopy, D. E. Bjorling, C. M. Vezina

This paper identifies an aging-related shift in procollagen 1A1 production from myofibroblasts to fibroblasts in the prostate of aging dogs.

PLOS ONE · Jul 29, 2020Peer-reviewedPathology

Factors Driving Unique Urination Phenotypes of Male and Female 9-week-old C57BL/6J Mice

H. M. RUETTEN, K. A. Wegner, H. L. Zhang, P. Wang, J. K. Sandhu, S. K. Sandhu, B. R. Mueller, Z. Wang, J. Macoska, R. E. Peterson, D. E. Bjorling, W. A. Ricke, P. C. Marker, C. M. Vezina

This study evaluates how prostate mass and testosterone affect voiding function in male and female mice, finding that testosterone, not prostate size, primarily drives sex differences in urinary function.

bioRxiv · May 27, 2019Physiology