Author

K. C. Berrett

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 3 Approach:experimental assay· 3 Organism:human· 3 Research Focus:cell type annotation· 2 Research Focus:chromatin & epigenetic regulation· 2 Assay:bulk RNA-seq· 1 Assay:ChIP-seq· 1 Research Focus:clustering· 1

Frequent Co-Authors

J. Gertz· 3 shared J. M. Vahrenkamp· 2 shared S. Arnesen· 2 shared S. Oesterreich· 2 shared Z. Blanchard· 2 shared Z. Li· 2 shared A. C. Beck· 1 shared A. J. Butterfield· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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3 shared papersCancer Biology

J. Gertz

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Author profile →

3 papers

Recent·Most cited

A breast cancer patient-derived xenograft and organoid platform for drug discovery and precision oncology

K. P. Guillen, M. Fujita, A. J. Butterfield, S. D. Scherer, M. H. Bailey, Z. Chu, Y. S. DeRose, L. Zhao, E. Cortes-Sanchez, C.-H. Yang, J. Toner, G. Wang, Y. Qiao, X. Huang, J. A. Greenland, , A. L. Welm

The paper develops patient-derived xenografts and organoids for breast cancer to enable drug screening and precision oncology, showing efficacy of an FDA-approved drug in a clinical case.

bioRxiv · Feb 28, 2021 · PreprintCancer Biology

Estrogen receptor alpha mutations in breast cancer cells cause gene expression changes through constant activity and through secondary effects

S. Arnesen, Z. Blanchard, M. M. Williams, K. C. Berrett, Z. Li, S. Oesterreich, J. K. Richer, J. Gertz

This study shows that ER mutations in breast cancer cells cause gene expression changes through constant activity and secondary effects, not just ligand independence.

bioRxiv · Apr 8, 2020Cancer Biology

Estrogen-independent molecular actions of mutant estrogen receptor alpha in endometrial cancer

Z. Blanchard, J. M. Vahrenkamp, K. C. Berrett, S. Arnesen, J. Gertz

This study investigates estrogen-independent actions of the D538G ESR1 mutation in endometrial cancer, showing altered genomic binding, gene expression, and chromatin accessibility compared to wildtype ESR1.

bioRxiv · Jul 3, 2019Cancer Biology