Author

K. C. V. Subbaiah

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 4

Signature Assays, Organisms & Methods

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Organism:mouse· 4 Assay:bulk RNA-seq· 3 Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Approach:experimental assay· 3 Research Focus:gene regulatory network inference· 3 Organism:human· 3 Research Focus:cancer & tumor microenvironment· 1

Frequent Co-Authors

J. Wu· 4 shared P. Yao· 4 shared W. H. W. Tang· 3 shared C. Yan· 2 shared F. Jiang· 2 shared O. Hedaya· 2 shared A. Mohan· 1 shared D. Mickelsen· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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4 shared papersPathology

J. Wu

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4 papers

Recent·Most cited

FAM210A Regulates Mitochondrial Translation and Maintains Cardiac Mitochondrial Homeostasis

J. Wu, K. C. V. Subbaiah, O. Hedaya, S. Chen, J. Munger, W. H. W. Tang, C. Yan, P. Yao

This study identifies FAM210A as a regulator of mitochondrial translation and cardiac mitochondrial homeostasis, showing its deficiency leads to heart failure and its overexpression rescues cardiac function in ischemic heart disease.

bioRxiv · May 20, 2023Pathology

FAM114A1 Influences Cardiac Fibrosis by Regulating Angiotensin II Signaling in Cardiac Fibroblasts

K. C. V. Subbaiah, J. Wu, W. W. Tang, P. Yao

This study identifies FAM114A1 as a regulator of cardiac fibrosis by modulating Ang II signaling in cardiac fibroblasts, showing its role in fibrosis and potential as a therapeutic target.

bioRxiv · Feb 20, 2021 · PreprintPathology

MicroRNA-574 Regulates FAM210A Expression and Influences Pathological Cardiac Remodeling

J. Wu, K. C. V. Subbaiah, F. Jiang, O. Hedaya, A. Mohan, T. Yang, K. Welle, S. Ghaemmaghami, W. H. W. Tang, E. Small, C. Yan, P. Yao

This study identifies miR-574 as a regulator of FAM210A, which influences mitochondrial protein expression and cardiac remodeling in heart failure.

bioRxiv · Jan 9, 2020Pathology

EPRS Regulates Proline-rich Pro-fibrotic Protein Synthesis during Cardiac Fibrosis

J. Wu, K. C. V. Subbaiah, L. H. Xie, F. Jiang, D. Mickelsen, J. R. Myers, W. H. W. Tang, P. Yao

This study identifies EPRS as a key regulator of proline-rich pro-fibrotic gene translation in cardiac fibrosis, showing its role in promoting fibrosis and identifying potential therapeutic targets.

bioRxiv · Sep 20, 2019Pathology